This is how professor Björn Hammarskjöld believes Moderna's COVID vaccine caused Nicholas to develop a heart inflammation - which paved the way for a bacterial infection
This is how professor Björn Hammarskjöld introduces himself:
”I am an assistant professor of pediatrics at Strömstad Academy, a former chief physician in pediatric medicine, and I hold a Bachelor with 2 grades in microbiology and 4 grades in biochemistry. Subsequently, I earned an old-fashioned Licentiate in Biochemistry from Stockholm University (1971) with a grade of Laudatur.
I worked as a Postdoc in molecular biology at the State University of New York at Buffalo, Buffalo, NY, USA, where I worked on the HIV envelope protein from 1988 to 1989. I have over 10 years of laboratory experience working with RNA, viruses, bacteria, and DNA manipulation.
Clinically, I have worked as a physician since 1977, primarily in pediatric medicine but also in general medicine and infectious diseases until my retirement in 2009.
I have authored a total of 22 scientific articles listed in PubMed, the major database in medical literature, and an additional 55 articles in other media.”
I came into contact with Hammarskjöld (yes, they are related) at the beginning of 2023. Since then, we have had a dialogue about Nicholas's medical situation. It was thanks to Björn Hammarskjöld's medical evaluation that prosecutor Helena Treiberg Claeson decided to initiate a preliminary investigation into involuntary manslaughter in September 2023.
The police have been informed of how, with the support of Professor Björn Hammarskjöld, I believe that Moderna's COVID vaccine killed Nicholas.
To argue the connection between Moderna's COVID vaccine and Nicholas's bacterial endocarditis and sepsis, one can summarize that the vaccine induced an inflammatory reaction, which damaged the tissue around his heart valve prosthesis.
Such inflammation can increase the permeability of blood vessels, making it easier for bacteria to penetrate and infect the tissue.
Together with potential immunosuppression, a vulnerable environment for bacterial infection was created, which may have led to the bacterial endocarditis and subsequent sepsis that led to Nicholas's death.
The combined effect of a strong inflammatory response, increased vascular permeability, potential immunosuppression, and an existing heart valve prosthesis provides a credible biological mechanism for how the vaccine may have contributed to Nicholas's condition.
1. Immune System Response and Inflammation
Pro-inflammatory Cytokines:
- mRNA vaccines induce the production of pro-inflammatory cytokines such as IL-1β and IL-6. These cytokines are important for creating an immune response but can also cause an excessive inflammatory reaction.
- This inflammation can damage tissues and create an environment where bacteria can more easily adhere and multiply. For individuals with prior heart surgery or heart valve prostheses, like Nicholas, these inflammatory reactions can be particularly problematic.
2. Vascular Permeability and Infection Risk
Increased Permeability:
- Pro-inflammatory cytokines can increase the permeability of blood vessels. This means that the vessel walls become more permeable, allowing bacteria to more easily enter the bloodstream and reach the heart valve.
- Inflammation in the vessel walls can lead to small injuries and an increased risk of bacterial adhesion and colonization. This is particularly relevant for individuals with heart valve prostheses, where damage to the tissue around the prosthesis can create ideal conditions for bacterial infection.
3. Underlying Medical Conditions
Heart Valve Prosthesis:
- Nicholas had a biological heart valve prosthesis (homograft conduit), which in itself increases the risk of bacterial infections such as endocarditis. Prostheses can be predisposing factors for bacterial colonization during inflammatory conditions.
- Inflammation caused by the vaccine's immune response may have damaged the tissue around the prosthesis and created attachment points for bacteria.
4. Specific Conditions and Disease Timeline
Timeline of Nicholas's Illness:
Nicholas received his second dose of Moderna's COVID vaccine on October 4, 2021. He developed a fever of 40 degrees Celsius the evening after the vaccination, indicating a strong immune response.
- Throughout November, he was tired and lethargic, which may indicate heart inflammation. This does not necessarily have more symptoms and requires more than the standard check-up that was done on the heart at the end of October to detect.
- Two months after the second dose, in early December 2021, he developed bacterial endocarditis and sepsis.
- This timeline is critical as it shows there was a significant period of inflammatory activity that may have created a vulnerability to bacterial infection. It is especially important to note that heart inflammation can be asymptomatic.
5. Microbiological Conditions
Spike Proteins and immunosuppression:
- Hammarskjöld argues that the spike protein, which Moderna's vaccine caused cells to express, can cause immunosuppression, reducing the body's ability to fight bacterial infections. This is supported by studies showing that strong inflammatory reactions can negatively affect the efficiency of the immune system.
- This immunosuppression, combined with an already existing prosthesis, may have created an environment that favored the development of bacterial endocarditis.
- The role of cytokines in the immune response to the spike protein is to draw immuncells such as T-cells to the infected area. Reactions from the T-cells can create attachment points for bacteria and holes that bacteria can penetrate into the bloodstream.
Image: Björn Hammarskjöld
References and Links:
1. Karlstad, Ø., Hovi, P., Husby, A., Härkänen, T., Selmer, R. M., Pihlström, N., Hansen, J. V., Nohynek, H., Gunnes, N., Sundström, A., Wohlfahrt, J., Nieminen, T. A., Grünewald, M., Gulseth, H. L., Hviid, A., & Ljung, R. (2022). SARS-CoV-2 Vaccination and Myocarditis in a Nordic Cohort Study of 23 Million Residents. JAMA Cardiology, 7(6), 600-612. https://doi.org/10.1001/jamacardio.2022.0583
2. WHO. (2021). GACVS guidance: myocarditis and pericarditis after COVID-19 mRNA vaccines. https://www.who.int/news/item/09-07-2021-gacvs-guidance-myocarditis-pericarditis-covid-19-mrna-vaccines
3. Garcia, G., Naveja, A., Bell, J., McBride, T., Waddell, J., & Wood, C. (2023). Cytokinopathy with aberrant cytotoxic lymphocytes and profibrotic macrophages in severe COVID-19. Science Immunology. https://www.science.org/doi/10.1126/sciimmunol.adh3455
4. Di Domenico, E. G., Cavallo, I., Bordignon, V., Prignano, G., Sperduti, I., Gurtner, A., Trento, E., Toma, L., Pimpinelli, F., Capitanio, B., & Ensoli, F. (2018). Inflammatory cytokines and biofilm production sustain Staphylococcus aureus outgrowth and persistence: a pivotal interplay in the pathogenesis of Atopic Dermatitis. Scientific Reports, 8(1), 9573. https://www.nature.com/articles/s41598-018-27421-1
5. Aikawa, T., Hatano, M., Yamanaka, S., & Aonuma, K. (2022). Non-infectious endocarditis and myocarditis after COVID-19 mRNA vaccination. European Heart Journal Case Reports, 6(1), ytab533. https://academic.oup.com/ehjcr/article/6/1/ytab533/6493260
6. Heymans, S., & Cooper, L. T. (2021). Myocarditis after COVID-19 mRNA vaccination: clinical observations and potential mechanisms. Nature Reviews Cardiology. https://www.nature.com/articles/s41569-021-00662-w